Zebrafish, C. elegans and Human Polycystic Kidney Disease
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Lyman Gingerich, Jamie S.
Meyer, Samantha
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Abstract
The focus of this project is to identify
the downstream effects of mutations in
the causative genes (PKD1 and PKD2) of
polycystic kidney disease (ADPKD).
ADPKD is responsible for 5% of all endstage
renal disease and is characterized
by large, fluid-filled cysts forming in
kidney tubules and collecting ducts that
disrupt the normal functioning of the
kidneys. It is currently thought that
primary cilia (1) and different signaling
pathways may play a major role in the progression of ADPKD.
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Color poster with text, graphs, charts, and images.
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University of Wisconsin--Eau Claire Office of Research and Sponsored Programs